Selasa, 01 Agustus 2017

Constipation



Constipation

DIGESTIVE DISORDERS- Constipation
Elimination of waste is equally as important as intake of healthy foods. That is why care is taken for proper evacuation right from birth. A newborn is watched for passing the meconium (first stool) within 36 hours of birth. Even in breast-fed infants, the passing of stool, at least once in three days, is essential.
For real internal health, good digestion and regular bowel movements/habits are very essential. The bile pigments and other wastages are usually excreted through faeces. So, even during fasting, small quantity of faeces will be passed on indicating the importance of defecation.
Less than 3 bowel movements in a week are considered medically as ‘constipation’. But, generally, ‘normal’ varies from person to person, i.e., some would go for stool once in 3 days and they won’t feel any discomfort or difficulty and would feel that is normal.
Some need coffee or milk or plenty of water or jogging to induce the act of defecation in themorning. Some need beer, alcohol, cigarette or tobacco or betel nut for free stools. Many people read the newspaper, enjoy music, enjoy smoking and work out the day planner in the toilet. These attitudes have brought about the closet trend to make bathrooms as glamour rooms with sophisticated accessories equal to living rooms. These things should be avoided and one should concentrate on passing the stool and leaving the toilet as early as possible.
Nowadays, people cannot digest food well and evacuate it effectively due to various reasons like bad quality of food, fast foods, drugs, habits, mental strain, etc. Also the tongue is given more importance than the stomach. For constipated people, constipation may be the first failure or upset of the day.
About constipation – Constipation is infrequent and inadequate defecation without satisfaction. Simply, it means difficulty in passing stool either in evacuating or act or time or in getting satisfaction. Some find difficulty in passing stool and take a long time in the toilet, due to hardness, bulkiness and dryness of the stool. Some may go many times to the toilet, but they may not succeed in emptying their bowels even though the stool seems to be soft. They constantly feel the urge and crumbling of stool in the rectum. All these will lead to lack of concentration and various health problems. Mostly, constipation occurs as a functional disorder, but people, due to carelessness and ignorance, make it a chronic problem. Also, many resort to self-medication and complicate it without finding the cause.
Incidence – Most people suffer from constipation at some time or the other. Old people suffer more commonly. At least 5–10 per cent of the population suffers and lives with constipation. Constipation can occur in all age groups, including newborn. Females suffer more commonly than males. They suffer especially during pregnancy and after delivery and develop fissures or piles. One who deviates more from nature in diet, living & sitting habits will usually suffer from higher incidences of constipation.
Pathophysiology – Easy defecation is close to good digestion. Good digestion starts from the mastication of food in the mouth. Our stomach and duodenum work more with digestion of food particles with gastric acid and bile. Coiled small intestines and large intestines work more with absorption. Nutrients and water are absorbed in the intestines as food passes slowly through this coiled long route with swaying motion of the intestines. As food loses nutrients and water while passing through these roller-coaster intestines, it becomes solid stool. In case of dehydration or increased absorption of water, the stool becomes dry and hard and gets stuck/impacted to the walls of the intestines to resist further movement, causing constipation. Water absorption from digested particles in the intestines plays a major role in constipation. The longer it remains, the more it becomes hard and dry.
Types of constipation – Constipation can be classified as acute and chronic depending upon the duration of suffering. Acute constipation with severe abdominal pain should be treated immediately to rule out any obstruction or impaction to avoiding serious consequences. Constipation can be classified into three types in the following manner:
Atonic constipation – Functional constipation occurring commonly due to lesser intake of water, less fibre content and irregular meals, vitamin deficiency, temporary and after effects of laxatives and purgatives.
Spastic constipation – due to irritability of intestines from foods, medicines, stress and strain, nervous disorders, etc.
Obstructive constipation – due to tumours or fecal impaction arresting the pathway
Causes – Human beings are often get constipated due to evolutionary changes, familial constitution and tension in the present world. Most times, the causes for constipation persist as hidden and no obvious reason can be traced and it becomes ‘non-specific’. It may be usually due to
Habits
  • Failure in developing habits
  • Controlling or suppressing the urge for a prolonged time
  • Making changes in routines
  • Habits of sedentary, being busy, long distance travel and alcohol
  • Irregular food habits at irregular time
Food & water
  • Low fibre food, highly-refined food, food with more spices and chillies
  • Less intake of water and dehydration caused by carbonated drinks and alcohol may cause constipation
  • Hereditary – Constipation is often found to run in families.
  • Anatomical – Any malformation/abnormality in anus, rectum and intestines
  • Physiological – Pregnancy, delivery and heavy exercises can cause constipation.
Pathological
  • Digestive tract – Anal fissure, fistula, prolapse, pelvic and rectal tumours/cancer, liver and portal vein disorders, large megacolon, Crohn’s disease, intestinal obstruction / stricture / adhesions, etc., can cause constipation.
  • Nervous disorders – Multiple sclerosis, Parkinson’s disease, stroke, spinal cord injuries, sluggish intestines, paralysis, etc.
  • Hormonal – diabetes, hypothyroidism, hypoparathyroidism
  • Psychological – Worries and mental strain can also cause havoc and lead to constipation
  • Environmental – Unfamiliar environments and a hot summer can lead to constipation
Medications – Drugs used in irritable bowel syndrome, diarrhoea, painful conditions, psychological conditions (antidepressants) and neurological conditions will often cause constipation. Supplements like iron and calcium, abuse or regular use of antacids and laxatives can also cause constipation.
Associated symptoms of constipation – Constipation as a symptom (not a disease) will not bother anyone in the initial days, but as time goes, it causes loss of appetite with abdominal discomfort/pain and bloating. While straining for passing dry hard stool, it may cause pain in the rectum/anus with occasional bleeding. Constipation usually accompanies belching, burning eructations, gas trouble, nausea, vomiting, headache, bad breath, weight loss, tiredness, lack of concentration, etc.
Diagnosis and investigations – Diagnosis is important to rule out any serious illnesses like obstruction, tumour / cancer and deformities. So investigation for the cause/disease should be properly done. Common tests which help in finding the cause are digital examination (finger palpation) / proctoscope / sigmoidoscope / colonoscope / endoscopy / barium enema X-ray (if necessary MRI or CT scan) / colorectal transit study / biopsy / stool examination / routine blood investigations with thyroid profile and calcium levels to rule out every possibility of presence of disease.
Preventive measures – The following procedures will surely help prevent/avoid recurrence of constipation
  • Move more towards greens and nature.
  • Don’t be sedentary and sit idle for a long time.
  • Eat fibre-rich diet for good digestion and easy defecation
  • Take plenty of water
  • Take plenty of fruits/fruit juices
  • Follow regular bowel habits and don’t be in a hurry in the toilet
  • Always answer the morning call/urge
  • Try to lead life without tension
  • Indian style closet is always better than Western since squatting is the apt posture to compress the abdomen to increase pressure for easy evacuation
Precautions – Avoid the following things to avoid constipation and complications:
  • Suppressing bowel movements or urge
  • Self-medication with laxatives
  • Continuous use of laxatives / stool softeners / enema
  • Don’t go for purgatives to clean stomach (if you are cleaning naturally everyday, it is unnecessary for shut down cleaning)
  • Straining severely for defecation
  • Spicy foods, meat and processed foods which need more time to digest
  • Carbonated drinks, alcohol and smoking
Self care and management – Taking
  • Lemon, honey, guava, grapes will facilitate easy digestion and defecation
  • Paraffin oil, olive oil, castor oil will facilitate passing of stool by lubrication
  • Vegetables, green leaves, cabbage, carrot, beans, grains, cereals can ease stool
Complications of constipation – One should be very careful with constipation or otherwise it may lead to complications like piles, fissures and prolapsed anus/rectum. Straining at stool may often cause hernia. In case of patients suffering with coronary artery diseases (heart complaints), straining may also cause palpitations, fainting and sweating.
Treatment for constipation – This commonest complaint is very commonly left untreated or self-treated with drugs available across the counter. This attitude leads to recurrence of constipation and complications. Surely, it needs the advice/diagnosis of a doctor, even though self-care, diet care and lifestyle changes works good. Morning results of laxatives and immediate result of enemas often compel every doctor to prescribe it even for the mild constipation of every patient in a hurry. Nowadays different types of laxatives are available in the market like bulk-forming, saline laxative, stimulants, etc. Stool softeners and lubricants are sometimes prescribed, depending upon the causes. Impacted stool can be treated with medicines and stool softeners, but intusseuption (inner riding of intestines into another part of intestine) and obstruction due to growth will often require surgical correction.
Homeopathic approach – Best results are often shown when one reverts to nature in foods and habits rather than treatments. Keeping soft stool is the key to success in the management of constipation. The mode of treatment usually varies depending upon the cause, severity, and duration. Treatment mode / method may also vary according to the doctor and system. In Homeopathy, constipation is well treated with total care for its cause and associated symptoms.
Homeopathy always gives importance to the general symptom than the disease symptoms. Stool is a general symptom i.e., it affects the person as a whole. So, Homeopathy usually gives importance to stool habit like thirst, sweat, sleep, appetite, urine, menses, etc., while treating any disease. Rediscover of bowel health can be attained earlier compared to the use of laxatives / stool softeners / enemas.
Constipation may be the leader of diseases in some cases. For example, constipated people usually have headache. For this, instead of giving painkillers or vasodilator for headache, clearing constipation gives cure once for all, unlike other temporary managements. Also, every action has its own reaction. If you sleep with sleeping pills today, then tomorrow night will be a sleepless one. Likewise, giving laxative instead of treating the cause will lead to more and more constipation in future. Any supplement or anti-pathy mode of approach will often lead to dependency throughout life. So, to remind our body to function on its own, Homeopathy is the best way.
Homeopathic medicines commonly used in the case of constipation are Acid Nit, Aesculus Hip, Aloes, Alumina, Alumen, Ammon mur, Bryonia, Calc carb, Capsicum, Carboveg, Causticum, Chamomilla, Collinsonia, Conium, Graphites, Hamemelis, Hepasulf, Hydrastis, Hypericum, Ficus.R, Ignatia, kali carb, Lachesis, Lycopodium, Mag Phos, Mag Mur, Muriatic acid, Nat Sulp, Nux Vom, Opium, Paeonia, Phosphorus, Plumbum met, Podophyllum, Pulsatilla, Ratahnia, Ruta, Sanicula, Sulphur, Tarentula, Thuja, etc. These Medicines should be taken under the advice and diagnosis of a qualified Homeopath.


for new hope

Dr. S. Chidambaranathan, BHMS, MD (Homeo)
Laxmi Homeo Clinic
24 E. New Mahalipatti Road
Madurai, TN 625 001
India

Tel:  +91-452-233-8833 | +91-984-319-1011 (Mob)
Fax: +91-452-233-0196
E-mail:  drcheena@yahoo.com
www.drcheena.com / www.drcheena.in


 

(Disclaimer: The contents of this column are for informational purpose only. The content is not intended to be a substitute for professional healthcare advice, diagnosis, or treatment. Always seek the advice of healthcare professional for any health problem or medical condition.)


How Do Anti Convulsants Work For Neuropathy


Today's post from painhq.org (see link below) looks at a common treatment for neuropathic pain and that is anti-convulsants or anti-epilepsy drugs. Many people living with neuropathy are prescribed these drugs after anti-depressants have failed and they are generally a pre-cursor to opiates (if they don't work for you). It is wise to consult carefully with your doctor (especially with Lyrica)depending on which anti-convulsant is prescribed and what the cause of your neuropathy is, as the side effects can be significant and for certain groups these drugs haven't been proven to work at all. It is a useful article in that it explains what they are and how they work and very often patients are not given this information by their doctors because it's assumed the patient will have difficulty understanding the science behind the drug's working process.


Anticonvulsants 
No author or date available

 
What are Anticonvulsants?

Anticonvulsants are a group of drugs that were designed to help manage seizures, but have since been used in the treatment and management of neuropathic pain. It is a fairly large and diverse family of drugs, sometimes referred to as ‘anti-seizure medications’ or ‘antiepileptics’. These drugs are thought to work through a number of different mechanisms: some may block different neurotransmitters; others may affect nerve signalling and ‘firing’ through binding to different receptors; altering ion channels in the brain; or ‘stabilizing’ some of the nerve cell membranes to ‘quiet’ pain signalling. On balance, it is thought that through these various mechanisms anticonvulsants affect pain communication pathways for patients with neuropathic pain.

How do they work?

Anticonvulsants were first used in pain management because it was thought that the nature of pain was somewhat similar to that of epileptic seizures – too much nerve cell firing. A number of these drugs have been shown to block the signals from damaged neurons which would normally communicate pain within the body.

What kinds are there? 

 
Gabapentin (Neurontin): Gabapentin works by binding to the calcium channels in neurons. These calcium channels help communicate pain within the body and, when blocked, help to dull the signal. Gabapentin is one of the only drugs used to treat neuropathic pain that is solely metabolized through the kidney (most are metabolized through the liver). It is considered one of the ‘first line’ medication treatments for most neuropathic pain syndromes. It is often used for treating post-herpetic neuralgia and diabetic neuropathy. 


Pregabalin (Lyrica)
: Developed as a more potent follow-up to Gabapentin, Pregabalin has been prescribed for postherpetic neuralgia, diabetic peripheral neuropathy and central neuropathic pain. Pregabalin is also associated with a lower risk for dependency and potential abuse. 

 
Carbamazepine (Tegretol)
/ Oxcarbazepine (Trileptal): Carbamazepine works by binding to sodium channels in neurons and limiting pain signals. Used It is frequently used to treat trigeminal neuralgia, but also diabetic neuropathy and potentially other forms of neuropathic pain (though more research is necessary). 


Valproate (Sodium valproate, valproic acid)
: Valproate helps block calcium channels and increases the levels of GABA in the brain. The calcium channels help to communicate pain while GABA helps to black dull pain signals in the brain by blocking communication channels and affecting nerve transmission. More evidence is needed to determine the use of Valproate in the treatment of neuropathic pain. 


Lamotrigine (Lamictal): Lamotrigine helps to block sodium channels and helps to regulate signals between neurons. Used it is sometimes used in the treatment of trigeminal neuralgia, diabetic neuropathy and central neuropathic pain. There are increased risks of side effects in woman using Lamotrigine, which also poses certain risks for pregnancies. 


Topiramate (Topamax): Topiramate has a number of mechanisms of action including sodium channels, calcium channels, GABA receptors, AMPA receptors and carbonic anhydrases. More evidence is needed to determine the use of Topiramate in the treatment of neuropathic pain.


Levetiracetam (Keppra): Levetiracetam works by binding to calcium channels in neurons, though it’s mechanisms aren’t fully understood. It can be used in the treatment of peripheral neuropathic pain. 


Lacosamide (Vimpat): Lacosamide works by binding to sodium channels in neurons, which prevents them from firing. Lacosamide also targets the cell which have been active for a longer period of time; in other words, damaged, over-active nerve cells that are sending pain signals (versus healthy cells). Lacosamide is used in the treatment of diabetic peripheral neuropathy.

What kind of Anticonvulsant is most effective for you?

Gabapentin (0)

Pregabalin (0)

Carbamazepine (0)

Valproate (0)

Lamotrigine (0)

Lacosamide (0)

Levetiracetam (0)

Topiramate (0)

Related evidence

Hearn L, Derry S, Moore RA. Lacosamide for neuropathic pain and fibromyalgia in adults. Cochrane Database Syst Rev. 2012 Feb 15;2:CD009318. doi: 10.1002/14651858.CD009318.pub2.
Moore RA, Straube S, Wiffen PJ, Derry S, McQuay HJ. Pregabalin for acute and chronic pain in adults. Cochrane Database of Systematic Reviews 2009, Issue 3. Art. No.: CD007076. DOI: 10.1002/14651858.CD007076.pub2
Moore R, Wiffen PJ, Derry S, Toelle T, Rice AS C. Gabapentin for chronic neuropathic pain and fibromyalgia in adults. Cochrane Database of Systematic Reviews 2014, Issue 4. Art. No.: CD007938. DOI: 10.1002/14651858.CD007938.pub3

Price MJ. Levetiracetam in the treatment of neuropathic pain: three case studies. Clin J Pain. 2004 Jan-Feb;20(1):33-6.

Wiffen PJ, Derry S, Moore RA, Aldington D, Cole P, Rice AS, Lunn MP, Hamunen K, Haanpaa M, Kalso EA. Antiepileptic drugs for neuropathic pain and fibromyalgia - an overview of Cochrane reviews. Cochrane Database Syst Rev. 2013 Nov 11;11:CD010567. doi: 10.1002/14651858.CD010567.pub2.

Wiffen PJ, Derry S, Lunn MPT, Moore R. Topiramate for neuropathic pain and fibromyalgia in adults. Cochrane Database of Systematic Reviews 2013, Issue 8. Art. No.: CD008314. DOI: 10.1002/14651858.CD008314.pub3

http://www.painhq.org/learning/knowledge-base/category/treatments/traditional-medicine-and-surgery/anticonvulsants

NICOTINE WITHDRAWAL REDUCES RESPONSE TO REWARDS ACROSS SPECIES



Cigarette smoking is a leading cause of preventable death worldwide and is associated with approximately 440,000 deaths in the United States each year, according to the U.S. Centers for Disease Control and Prevention, but nearly 20 percent of the U.S. population continues to smoke cigarettes. While more than half of U.S. smokers try to quit every year, less than 10 percent are able to remain smoke-free, and relapse commonly occurs within 48 hours of smoking cessation. Learning about withdrawal and difficulty of quitting can lead to more effective treatments to help smokers quit.

In a first of its kind study on nicotine addiction, scientists measured a behavior that can be similarly quantified across species like humans and rats, the responses to rewards during nicotine withdrawal. Findings from this study were published online on Sept. 10, 2014 in JAMA Psychiatry.

Response to reward is the brain's ability to derive and recognize pleasure from natural things such as food, money and sex. The reduced ability to respond to rewards is a behavioral process associated with depression in humans. In prior studies of nicotine withdrawal, investigators used very different behavioral measurements across humans and rats, limiting our understanding of this important brain reward system.
Using a translational behavioral approach, Michele Pergadia, Ph.D., associate professor of clinical biomedical science in the Charles E. Schmidt College of Medicine at Florida Atlantic University, who completed the human study while at Washington University School of Medicine, Andre Der-Avakian, Ph.D., who completed the rat study at the University of California San Diego (UCSD), and colleagues, including senior collaborators Athina Markou, Ph.D. at UCSD and Diego Pizzagalli, Ph.D. at Harvard Medical School, found that nicotine withdrawal similarly reduced reward responsiveness in human smokers -- particularly those with a history of depression -- as well as in nicotine-treated rats.
Pergadia, one of the lead authors, notes that replication of experimental results across species is a major step forward, because it allows for greater generalizability and a more reliable means for identifying behavioral and neurobiological mechanisms that explain the complicated behavior of nicotine withdrawal in humans addicted to tobacco.

"The fact that the effect was similar across species using this translational task not only provides us with a ready framework to proceed with additional research to better understand the mechanisms underlying withdrawal of nicotine, and potentially new treatment development, but it also makes us feel more confident that we are actually studying the same behavior in humans and rats as the studies move forward," said Pergadia.

Pergadia and colleagues plan to pursue future studies that will include a systematic study of depression vulnerability as it relates to reward sensitivity, the course of withdrawal-related reward deficits, including effects on relapse to smoking, and identification of processes in the brain that lead to these behaviors.
Pergadia emphasizes that the ultimate goal of this line of research is to improve treatments that manage nicotine withdrawal-related symptoms and thereby increase success during efforts to quit.
"Many smokers are struggling to quit, and there is a real need to develop new strategies to aid them in this process. Therapies targeting this reward dysfunction during withdrawal may prove to be useful," said Pergadia.


Chronic Inflammatory Demyelinating Polyneuropathy CIPD More Than Meets The Eye


Today's post from hillandponton.com (see link below) has a long title, describing a severe form of neuropathy called chronic inflammatory demyelinating polyneuropathy but in fact, the content applies to practically everyone who suffers from neuropathy and in that respect, is a useful description of what neuropathy entails. The title is also a long-winded description of the form of nerve damage that many people suffer from but is more often called peripheral neuropathy, or just neuropathy. CIPD involves damage to the myelin sheath insulating the nerves but this also applies to most cases of peripheral neuropathy. However, it also suggests that the standard protocol for CIPD neuropathy treatment involves just three components: IVIG (intravenous immunoglobulin); Corticosteroids and plasma exchange. That this is misleading is an understatement. Treatment for CIDP is much wider than that and is certainly not limited to those three options as most patients will know. It's difficult to see where the author has got her information from and from what angle she's approaching the subject but this article is an example of how you need to be careful not to jump to conclusions from just reading one source of information.


What is Chronic Inflammatory Demyelinating Polyneuropathy (CIDP)?
 March 6, 2017/in Veterans /by Brenda Duplantis, Accredited Claims Agent

Chronic inflammatory demyelinating polyneuropathy (CIDP) is a neurological disorder characterized by gradually increasing sensory loss and weakness associated with the loss of deep tendon reflex in the legs and arms. The hallmark of a peripheral nerve disease is loss of ankle reflex. This is when your doctor hits your Achilles tendon and there is no reflex.

CIDP is a progressive motor and sensory neuropathy that is very painful and debilitating. CIDP is caused by damage to the covering of the nerves, called myelin.



The axon (nerve fiber) works like an electric wire. The myelin sheath around the axon is the insulation necessary for the nerve to conduct electrical impulses properly. In CIDP, myelin is attacked through very complex mechanisms. In such cases, the body sees the peripheral nerve as foreign and antibodies bind to the myelin and begin to break it down. In people with CIDP, this translates into symptoms. For example, if you want to move your finger, messages tell your brain to move the finger. Then electrical communication within your body tells your nerves and muscles to move the finger. When there is disruption in the myelin sheath, those messages are weakened. Your brain is telling your finger to move but the nerves are damaged and the movement is weakened, delayed, or absent. When the myelin sheath is completely damaged, the message is blocked and this is called paralysis.

The disease can be present in a person for years prior to diagnosis. Because it is a gradually progressing disorder and its symptoms may, at early stages, wax and wane, a definitive diagnosis may require invasive tests. However, a neurologist that has experience in this type of disease should be able to identify the gradual symptoms and rule out any other cause. Typically, a diagnosis of CIDP is based on the person’s symptoms. Common symptoms in CIDP patients include:

In extremities:

 
Pain
Tingling
Numbness
Weakness
Loss of deep tendon reflex
Foot drop
Difficulty walking (altered gait, stumbling)
Fatigue

In addition to the aforementioned symptoms, tests such as a nerve conduction study (NCS) and electromyography (EMG) – a diagnostic procedure to assess the health of muscles and the nerve cells that control them – may be administered to determine the extent of demyelinating disease. In demyelinating disease, there is damage to the lining of the nerves that are critical for electrical nerve conduction. This can be confirmed by EMG studies or nerve biopsy. A nerve biopsy is used to confirm inflammatory process in the patient’s nerve.

A spinal fluid analysis is another diagnostic test that helps determine if a patient has elevated protein with normal cell count, an abnormality found in CIDP patients. Finally, your doctor may order blood and urine tests to rule out other disorders that may cause neuropathy, such as diabetes, which is the number cause of peripheral neuropathy.

The therapy for CIDP includes these three primary protocols:

 
IVIG (intravenous immunoglobulin used to treat various autoimmune, infectious, and idiopathic diseases) – can aggravate kidney dysfunction, cardiovascular disease, cerebrovascular disease, and other.
Corticosteroids (oral prednisone, pulse oral dexamethasone, IV methylprednisolone) – often improve strength, are conveniently taken by mouth, and are inexpensive. Side effects, however, can limit long-term use.
Plasma exchange – a process by which some of the patient’s blood is removed and the blood cells returned without the liquid plasma portion of the patient’s blood. It may work by removing harmful antibodies contained in the plasma. Short-term relief.

If treated early, most CIDP people respond well to therapy that can reduce the damage to peripheral nerves and contribute to improved function and quality of life. If left untreated, 30% of CIDP patients will progress to wheelchair dependence.

https://www.hillandponton.com/cidp/

Senin, 31 Juli 2017

HOMOEOPATHIC REMEDIES FOR NERVE AFFECTIONS


A nerve is an enclosed, cable-like bundle of axons (nerve fibers, the long and slender projections of neurons) in the peripheral nervous system. A nerve provides a common pathway for the electrochemical nerve impulses that are transmitted along each of the axons to peripheral organs.
In the central nervous system, the analogous structures are known as tracts. Neurons are sometimes called nerve cells, though this term is potentially misleading since many neurons do not form nerves, and nerves also include non-neuronal Schwann cells that coat the axons in myelin.
Each nerve is a cordlike structure that contains many axons. Within a nerve, each axon is surrounded by a layer of connective tissue called the endoneurium. The axons are bundled together into groups called fascicles, and each fascicle is wrapped in a layer of connective tissue called the perineurium. Finally, the entire nerve is wrapped in a layer of connective tissue called the epineurium
HOMOEOPATHIC REMEDIES .
ACONITUM NAPELLUS 30- Suuden inflammation of the nerves due to anxiety or fear worse with noise and light
APIS MEL 30- Optic neuritis in the first stage
ARGENTUM NITRICUM 30- Inflammation of the nerves. Loss of control and want of balance anywhere- mental and physical
BELLIS PRENNIS 30- Results of injuries to nerves with intense soreness and intolerance to cold bathing
CARBONEUM SULPH 30- For atrophy of optic nerve and  optic disc
CHENOPODIUM AN. 30- Affections of auditory nerve . Hearing better for high pitched sounds. Comparative deafness to  the sound of voice. Burning in ears. Numbness of the auditory nerve
CIMCIFUGA RACEMOSA 30- Inflammation of nerves due to reflex disorders
CINNABARIS 3X –Pain in the ciliary nerve causing redness of the eyes, canthi and lids. Pain around the eyes to templates and orbit of the eyes
CYPRIPEDIUM 30- Nerves shortened by long illness or excessive tea or coffee drnking
GLONOINUM 30- Inflammation of nerves due to heat of sun. Better by motion and uncovering the head. Pulsations all over the body
HYPERICUM PERF. 3X- Crushing injuries to the nerve sheaths of the spine and other nerves causing tearing , burning and stinging pains. Slight paralysis caused by enlargement of nerves in the sacrum. Numbness of parts affected and constant drowsiness
KALI PHOS 6X – An excellent nerve tonic
LYCOPERSICUM ESCU. 30- Tingling along the right ulnar nerve
MAGNESIUM PHOS 12X- It is a remedy of nerve tension as exhibited in pulse. In severe nerve tension , the wrists also become tense. If it is in both wrists , it shows that the whole nervous system is involved. Three tablets given with hot water 2 hourly will remove the tension. Pain is on the right side, better by heat and pressure
NAPHTHALINUM 30- Paralysis of the optic nerve causing blindness and opacity of the cornea
PHOSPHORUS 200-Atrophy of the optic nerve with cataract and due to paralysis
RHUS TOX 30, HYPERICUM 30, KALMIA LAT. 30- Pain along the ulnar nerve , according to symptoms of the remedy
SAPONARIA OFF. 30- Affections of the 5th nerve. Pain or loss of sensation in the face, forehead, temple and eyes. Deviation of jaw